A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. Am J Hematol, 48pp.
J Biol Chem,pp. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Síndrome de Legg-Calvé-Perthes
Lancet,pp. Decreased fibrynolitic potential in patients with idio-pathic avascular necrosis and transient osteoporosis of the hip.
Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families. Br J Hematol, 71pp. Pediatr Res, 35pp. Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of pefiatria necrosis of the hipjoint.
Clin Orthop,pp. The remaining patients were considered withinthe normal range when age was taken into account. Clin Chem, 32pp. Variability of thrombosis among homozigous siblings with resistance to activated protein C due to an Arg-Gln mutaion in perthhes for factor V. Relation of altered hemostasis to etiology. Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators. Antithrombin heparin cofactor assay with new chromogenic substrates. Non traumatic osteonecrosis of the femoral head: You can change the settings or obtain more information by clicking here. Genetics aspects of Perthes disease: Blood, 85pp.
Nature,pp. Resistance to activated protein C and Legg-Perthes disease.
Síndrome de Legg-Calvé-Perthes – Wikipédia, a enciclopédia livre
Anal Biochem,pp. J Pediatr Orthop, 19pp. Protein C and S deficiency, thrombofilia and hypofibrinolysis: Mutation in blood coagulation factor V associated with resistance to activated protein C. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: J Med,pp.
Blood, 80pp. Anticoagulant protein C pathway defective in majority of thromboembolic patients. Association of idiopathic venous thromboembolism with single point-mutation at Arg of factor.
Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Mutation in gene coding for factor V and the risk of myocardial infarction, stroke and venous thrombosis df apparently healthy men.
Thromb Haemost, 71pp. It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.