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Find articles by Sun Hee Lee The injuries of inner ear were measured with hearing thresholds, . as seen by an increase in the ABR threshold in the furosemide mg/kg however, there were no significant differences among them. . Tan B. T. G., Lee M. M. G., Ruan R. Bone marrow-derived cells that. We look forward to welcoming you to the 44th Annual Meeting in noise and smoking, among other possibilities. Hwan Baek, MD; Hee Sung. Lee, Eun Jee; Kim, Hee Sun. Lee, Eun Jee Alcohol Consumption and Binge Drinking Among Young Adults Aged 20–30 Years in Lisbon, Portugal. Moutinho.
Of particular importance, patients that first complained of otologic symptoms only took longer to be finally diagnosed with nasopharyngeal cancer. Since ear fullness was only considered as a simple otologic symptom, the lack of endoscopic and histological examination may have delayed the diagnosis for nasopharyngeal cancer.
Clinical Manifestations of Aural Fullness
Thus, ear fullness should not be considered as a simple otologic symptom, and should be carefully examined along with other accompanying symptoms. In conclusion, in examining outpatients whose main complaint was ear fullness, we found that the predominant final diagnosis was Eustachian tube dysfunction, followed by otitis media with effusion and chronic otitis media.
These patients should be examined by otoscopy, nasal endoscopy, the Valsalva maneuver, and additional audiological tests. Since many of our patients could not be initially diagnosed, it is necessary to take a detailed history, as well as perform thorough examinations and sustained observations of patients in order to arrive at a final diagnosis.
Footnotes The authors have no financial conflicts of interest. Treatment of idiopathic sudden sensorineural hearing loss with antiviral therapy: Ann Otol Rhinol Laryngol. Effect of estrogen on eustachian tube performance. The effect of diabetes on sensorineural hearing loss.
Lee Sun Hee – Meet Him Among Them Lyrics
Cognitive decline and dementia in diabetes--systematic overview of prospective observational studies. Auditory brainstem responses in patients under treatment of hemodialysis. The effects of single and dual sensory loss on symptoms of depression in the elderly. We also enrolled age- and gender-matched normal volunteers 10 earsfor each patient. Extended high frequency pure tone audiometry was performed, and the mean hearing thresholds at 10, 12, 14, and 16 kHz of each tinnitus ear were compared with those of the 10 age- and sex-matched normal ears.
Results Of the 18 patients with tinnitus, 12 had significantly increased hearing thresholds at more than one of the four high frequencies, compared with the normal group.Reaction - LEE SUN HEE - Meet him among them
When we assessed results according to frequency, we found that 8 patients had decreased hearing ability at 10 kHz, 10 at 12 kHz, 8 at 14 kHz, and 4 at 16 kHz. Conclusion Some patients with tinnitus who have normal hearing below 8 kHz have decreased hearing ability at extended high-frequencies.
Thus, the proportion of patients with tinnitus who have normal hearing over the entire audible range is smaller than in previous reports. Severity of tinnitus is often found to increase with the degree of hearing loss and the pitch of tinnitus often coincides with the frequency of the lesion or is just below the precipitous edge of the lesion.
These findings demonstrate that damage to cochlear hair cells plays a crucial role in the pathogenesis of tinnitus 3 - 7.
However, tinnitus can also appear in deaf patients or those in which the auditory nerve has been surgically sectioned. Studies measuring changes in positron emission computed tomography CT associated with tinnitus have supported the hypothesis that tinnitus originates in the central nervous system. Thus, tinnitus does not develop from damaged hair cells, but is a phantom perception caused by plastic readjustments of the auditory center due to decreased afferent signals 8 - In other words, in patients with hair cell destruction within a specific frequency range, there is a decrease in signal transduction of the corresponding frequency from the acoustic nerve.
A Simple Model for Inducing Optimal Increase of SDF-1 with Aminoglycoside Ototoxicity
This reduced nerve input, which has a characteristic frequency in the hearing loss range, may result in a reduction of lateral inhibition at the dorsal cochlear nucleus or inferior colliculus. The reduced nerve input may also be due to the development of tinnitus caused by the hypersensitivity and hyperactivity in auditory neurons at the edge frequency 10 In addition, tinnitus may be caused or changed by somatosensory or somatic motor nerves other than the acoustic nerve, suggesting that tinnitus may occur in the dorsal cochlear nucleus 11 To prove that this ototoxicity mice model, in which SDF-1 is markedly increased, could be the appropriate model to study homing, we confirmed the changes in SDF-1 levels with increasing hearing thresholds based on the various conditions of one-shot ototoxicity.
Thereafter, we demonstrated an effective homing phenomenon in a mouse model with maximal increase in SDF-1 levels in the inner ear. The animals were 5 weeks old and weighed approximately 18—25 g.
The animals underwent cardiac perfusion with phosphate-buffered saline PBS: The temporal bones were dissected, and the bony shells of the cochlea and vestibule were removed in chloride-free physiological saline. Ototoxic Drug Administration The first injection for each mouse was given at the beginning of the daily light cycle. The mice in the sham control group received a subcutaneous injection of saline, followed by another tail vein injection of saline 30 min later.